References
1. Isolation and characterization of concanamycins A, B and C: H. Kinashi, et al.; J. Antibiot. 37: 1333 (1984). 2. Folimycin (concanamycin A), a specific inhibitor of V-ATPase, blocks intracellular translocation of the glyco- protein of vesicular stomatitis virus before arrival to the Golgi apparatus: M. Muroi, et al.; Cell Struct. Funct. 18: 139 (1993). 3. Folimycin (concanamycin A), an inhibitor of V-type H(+)-ATPase, blocks cell-surface expression of virus- envelope glycoproteins: M. Muroi, et al.; BBRC 193: 999 (1993). 4. Inhibitory effect of modified bafilomycins and concanamycins on P- and V-type adenosinetriphosphatases: S. Drose, et al.; Biochemistry 32: 3902 (1993). 5. Involvement of the vacuolar H(+)-ATPases in the secretory pathway of HepG2 cells: M. Yilla, et al.; J. Biol. Chem. 268: 19092 (1993). 6. Characterization of the ATPase activity of P-glycoprotein from multidrug-resistant Chinese hamster ovary cells: F.J. Sharom, et al.; Biochem. J. 308 (Pt2): 381 (1995). 7. Specific inhibitors of vacuolar type H(+)-ATPases induce apoptotic cell death: T. Nishihara, et al.; BBRC 212: 255 (1995). 8. Concanamycin A, the specific inhibitor of V-ATPases, binds to the V(o) subunit c: M. Huss, et al.; J. Biol. Chem. 277: 40544 (2002). 9. Nitric oxide production by the vacuolar-type (H+)-ATPase inhibitors bafilomycin A1 and concanamycin A and its possible role in apoptosis in RAW 264.7 cells: J. Hong, et al.; J. Pharmacol. Exp. Ther. 319: 672 (2006). 10. Degradation of oxidized proteins by autophagy during oxidative stress in Arabidopsis: Y. Xiong, et al.; Plant Physiol. 143: 291 (2007). 11. Inhibitors of the V0 subunit of the vacuolar H+-ATPase prevent segregation of lysosomal- and secretory- pathway proteins: J.A. Sobota, et al.; J. Cell Sci. 122: 3542 (2009). 12. Inhibitors of V-ATPase: old and new players: M. Huss, et al.; J. Exp. Biol. 212: 341 (2009).USBio References
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